Authors

Halie M. Rando, University of PennsylvaniaFollow
Adam L. MacLean, University of Southern California
Alexandra J. Lee, University of Pennsylvania
Ronan Lordan, University of Pennsylvania Perelman School of Medicine
Sandipan Ray, Indian Institute of Technology Hyderabad
Vikas Bansal, Deutsches Zentrum für Neurodegenerative Erkrankungen
Ashwin N. Skelly, University of Pennsylvania Perelman School of Medicine
Elizabeth Sell, University of Pennsylvania Perelman School of Medicine
John J. Dziak, Pennsylvania State University
Lamonica Shinholster, Mercer University at Macon
Lucy D’Agostino McGowan, Wake Forest University
Marouen Ben Guebila, Harvard T.H. Chan School of Public Health
Nils Wellhausen, University of Pennsylvania
Sergey Knyazev, Georgia State University
Simina M. Boca, Georgetown University Medical Center
Stephen Capone, St. George's University School of Medicine
Yanjun Qi, University of Virginia School of Engineering and Applied Science
Yo Son Park, University of Pennsylvania
David Mai, School of Engineering and Applied Science
Yuchen Sun, University of Virginia School of Engineering and Applied Science
Joel D. Boerckel, School of Engineering and Applied Science
Christian Brueffer, Institutionen för Kliniska Vetenskaper, Lund
James Brian Byrd, University of Michigan Medical School
Jeremy P. Kamil, LSU Health Sciences Center - Shreveport
Jinhui Wang, University of Pennsylvania Perelman School of Medicine
Ryan Velazquez, Azimuth1
Gregory L. Szeto, Allen Institute for Immunology
John P. Barton, University of California, Riverside
Rishi Raj Goel, University of Pennsylvania Perelman School of Medicine
Serghei Mangul, USC School of Pharmacy
Tiago Lubiana, Universidade de São Paulo
Anthony Gitter, University of Wisconsin School of Medicine and Public Health

Author ORCID Identifier

Halie M. Rando: 0000-0001-7688-1770

Adam L. MacLean: 0000-0003-0689-7907

Ronan Lordan: 0000-0001-9668-3368

Sandipan Ray: 0000-0002-9960-5768

Ashwin N. Skelly: 0000-0002-1565-3376

John J. Dziak: 0000-0003-0762-5495

Lamonica Shinholster: 0000-0001-6285-005X

Marouen Ben Guebila: 0000-0001-5934-966X

Nils Wellhausen: 0000-0001-8955-7582

Simina M. Boca: 0000-0002-1400-3398

Stephen Capone: 0000-0001-7231-1535

Joel D. Boerckel: 0000-0003-3126-3025

Christian Brueffer: 0000-0002-3826-0989

Jeremy P. Kamil: 0000-0001-8422-7656

Ryan Velazquez: 0000-0002-3655-3403

Gregory L. Szeto: 0000-0001-7604-1333

Tiago Lubiana: 0000-0003-2473-2313

Anthony Gitter: 0000-0002-5324-9833

Casey S. Greene: 0000-0001-8713-9213

Document Type

Article

Publication Date

9-1-2021

Publication Title

mSystems

Abstract

The novel coronavirus SARS-CoV-2, which emerged in late 2019, has since spread around the world and infected hundreds of millions of people with coronavirus disease 2019 (COVID-19). While this viral species was unknown prior to January 2020, its similarity to other coronaviruses that infect humans has allowed for rapid insight into the mechanisms that it uses to infect human hosts, as well as the ways in which the human immune system can respond. Here, we contextualize SARS-CoV-2 among other coronaviruses and identify what is known and what can be inferred about its behavior once inside a human host. Because the genomic content of coronaviruses, which specifies the virus’s structure, is highly conserved, early genomic analysis provided a significant head start in predicting viral pathogenesis and in understanding potential differences among variants. The pathogenesis of the virus offers insights into symptomatology, transmission, and individual susceptibility. Additionally, prior research into interactions between the human immune system and coronaviruses has identified how these viruses can evade the immune system’s protective mechanisms. We also explore systems-level research into the regulatory and proteomic effects of SARS-CoV-2 infection and the immune response. Understanding the structure and behavior of the virus serves to contextualize the many facets of the COVID-19 pandemic and can influence efforts to control the virus and treat the disease. IMPORTANCE COVID-19 involves a number of organ systems and can present with a wide range of symptoms. From how the virus infects cells to how it spreads between people, the available research suggests that these patterns are very similar to those seen in the closely related viruses SARS-CoV-1 and possibly Middle East respiratory syndrome-related CoV (MERS-CoV). Understanding the pathogenesis of the SARS-CoV-2 virus also contextualizes how the different biological systems affected by COVID-19 connect. Exploring the structure, phylogeny, and pathogenesis of the virus therefore helps to guide interpretation of the broader impacts of the virus on the human body and on human populations. For this reason, an in-depth exploration of viral mechanisms is critical to a robust understanding of SARS-CoV-2 and, potentially, future emergent human CoVs (HCoVs).

Keywords

COVID-19, genomics, review, viral pathogenesis

Volume

6

Issue

5

DOI

10.1128/mySystems.00095-21

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