Document Type

Article

Publication Date

3-1-2012

Publication Title

Journal of the Peripheral Nervous System

Abstract

Peripheral neuropathy is a common side effect of a number of pharmaceutical compounds, including several chemotherapy drugs. Among these are vincristine sulfate, a mitotic inhibitor used to treat a variety of leukemias, lymphomas, and other cancers, and bortezomib, a 26S proteasome inhibitor used primarily to treat relapsed multiple myeloma and mantle cell lymphoma. To gain insight into the mechanisms by which these compounds act, we tested their effects in zebrafish. Vincristine or bortezomib given during late embryonic development caused significant defects at both behavioral and cellular levels. Intriguingly, the effects of the two drugs appear to be distinct. Vincristine causes uncoordinated swimming behavior, which is coupled with a reduction in the density of sensory innervation and overall size of motor axon arbors. Bortezomib, in contrast, increases the duration and amplitude of muscle contractions associated with escape swimming, which is coupled with a preferential reduction in fine processes and branches of sensory and motor axons. These results demonstrate that zebrafish is a convenient in vivo assay system for screening potential pharmaceutical compounds for neurotoxic side effects, and they provide an important step toward understanding how vincristine and bortezomib cause peripheral neuropathy.

Keywords

Behavior, Chemotherapy, Development, Neuropathy, Proteasome inhibitor

Volume

17

Issue

1

First Page

76

Last Page

89

DOI

10.1111/j.1529-8027.2012.00371.x

ISSN

10859489

Rights

© 2012 Peripheral Nerve Society

Comments

Archived as published. Open access article.

Included in

Biology Commons

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